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While this might seem counterintuitive, studies in mice suggest that hypoxia could potentially be used as a therapy for mitochondrial dysfunction, with the researchers proposing that hypoxia could be beneficial owing to two different mechanisms: the first involving a reduction in O 2 tension, resulting in a decrease of reactive oxygen species (ROS), while the second involving activation of HIFs (Hypoxia-Inducible Factors), resulting in decreased production of ATP by OXPHOS and a shift to glycolysis (Jain et al., 2016, 2019

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10.1109/TBME.2017.2773463 IEEE Trans