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Minocycline, a broad-spectrum antibacterial tetracycline antibiotic, facilitates white matter repair by ameliorating BBB damage, upregulating angiogenesis and the expression of tight junction proteins, and finally promoting NG2-glia proliferation to maintain oligodendrocytes in rats [105]

Microglia, neurons, astrocytes, and endothelial cells can release proinflammatory cytokines and chemokines, modulating adhesion molecules and transmigration of activated immune cells into the brain parenchyma (Jickling et al., 2018)

The addition of BPC-157 and TB-500 theorizes a faster, more robust restructuring of the dermal-epidermal junction