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PROTACs recruiting the cereblon E3 ligase have successfully induced the forced degradation of more than 60 different proteins, associated with the development of a wide range of disorders, from cancer to neurological, immunological and viral diseases, with successful examples already in clinical trials 54,55,56,57,58

The process involves the selective sequestration of excess or impaired mitochondria in autophagosomes, which subsequently fuse with lysosomes, ultimately leading to mitochondrial degradation

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In human T cells, it induces FOXP3 protein expression and a suppressive phenotype but in contrast to vitamin C does not induce demethylation of TSDRs of the FOXP3 locus [10]