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Initially identified as a cofactor specifically interacting with the human immunodeficiency virus (HIV-1) Tat protein, an activator of HIV gene expression [33] , Tip60 was later recognized as a substrate-specific histone acetyltransferase with an evolutionarily conserved domain shared with yeast silencing factors [34]

More importantly, it provides the dual-action metabolic support discussed earlier

Under conditions of insulin resistance, hepatic lipid metabolic regulation becomes profoundly disrupted, characterized by persistent activation of fatty acid synthesis, suppression of fatty acid oxidation, and increased VLDL secretion, ultimately leading to hypertriglyceridemia and hepatic steatosis (25)