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We previously reported that reduced levels of activated hepatocyte growth factor and the c-Met receptor tyrosine kinaseboth of which are vital for maintaining cellular redox balancecombined with increased expression of GJ proteins in hepsin-deficient mice

Conversely, another subset of CD8 + T cells can augment vascular inflammation through the production of IFN and TNF, and worsen plaque instability by killing VSMCs and VECs 2,4