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The ability to increase T cell clonality in RRMM patients was reported for both anti-CD38 agents in clinical trials [44, 45]

As copper is a cofactor in tumor angiogenesis, and its high levels influence tumor growth [110], Cu agents potentially can be used for BRAFV600E mutation-positive malignancies treatment and tumors resistant to BRAFV600E and MEK1/2 inhibitors [111]