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World Health Organization Ivanova L, Fste CK, Uhlig S (2011) In vitro phase I metabolism of the depsipeptide enniatin B

However, the mechanism beneath the effect of GSH depletion-induced oxidative stress in RPE cells and the subsequent RPE damage is not well established

Additionally, the peptide apparently stimulated dermal keratinocyte proliferation, as evidenced by histological analysis of biopsies

Elesclomol-induced copper retention within mitochondria promoted oxidative stress and consequent ferroptosis in colorectal cancer cells via copper-transporting ATPase 1 (ATP7A) degradation and ROS accumulation ( Figure 1A )