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CPP-C16 conjugates inhibit NFAT-driven transcription and IL-2 expression in human T cells Having established that the CPP-C16 conjugates successfully inhibit the nuclear translocation of NFAT, we next sought to determine whether this cytosolic retention translates to a reduction in downstream NFAT-dependent transcriptional activity

Thymosin Alpha1: An Endogenous Regulator of Inflammation, Immunity, and Tolerance

The morphological manifestation of ferroptosis is distinguished by diminished mitochondrial cristae, atrophy of entire mitochondria, normal size of the nucleus, and intact cell membrane

[2] It functions primarily as an antioxidant, with intracellular concentrations reaching up to 20 mM in cells