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FIGURE 4 Deletion of endogenous H 2 S is critically involved in diabetes-induced cardiac damage, whereas exogenous H 2 S donor NaHS improves diabetic cardiomyopathy though inhibition of endoplasmic reticulum stress ( 2 S therapy (SG-1002) is capable of relieving HFD-induced cardiomyopathy via suppressing cardiac endoplasmic reticulum stress and restoring cardiac adiponectin levels ( 2 S donors are postulated to serve as novel cardioprotective agents for diabetic cardiomyopathy due to its strong suppression of endoplasmic reticulum stress-dependent apoptosis pathway in the heart

The landscape of bispecific T cell engager in cancer treatment