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Conclusions Intestinal barrier and bacterial-derived products seem to play an increasingly significant role in several chronic diseases

Through EMT, epithelial cells lose cell polarity, connections to the basement membrane and other epithelial phenotypes, and gain mesenchymal phenotypes such as increased migratory and invasive abilities, anti-apoptotic features and the capacity to degrade the extracellular matrix (79)

GLP-1 RAs, by addressing both insulin resistance and adiposity, occupy a mechanistically plausible position in this interface

Research in animal models shows enhanced collagen deposition and reduced healing time in injury sites treated with thymosin fragments [11]