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Together, increased intraplaque cellular death and dysfunctional efferocytosis as a result of G6PD-d may accelerate plaque growth, necrotic core formation and the development of an inflammatory plaque microenvironment 106,107 (Fig

This includes executive and visuospatial function, impaired short-term memory, language, and attention, contrasting to patients without the polymorphism and/or HCs (Mata et al., 2014

This could explain the genetic mechanism by which LC or LCLN enhances the studied hormones and subsequently improves spermatogenesis