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It has been shown that these two species play a role in inflammatory signaling, and inflammation has been shown to be involved in PD pathogenesis (Cunningham et al., 2008), while the higher levels of glycerophosphoinositol and glycerophosphoserine may cause defects in mitochondrial turnover (Lobasso et al., 2017)

1 2 "ApoE-deficient mice are a model of lipoprotein oxidation in atherogenesis"

Background studies published before 2010 and considered seminal or foundational were also included to provide the necessary background and context to the literature

At the same time, the important pathways and key genes involved in this study can provide new ideas for potato heavy metal tolerance breeding in the future