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Furthermore, at 2A -adrenergic receptors, DA functions as a biased ligand, preferentially signaling via G protein recruitment but not through -arrestin2, providing a new mechanism for DAs intracellular signaling at these receptors

Sansone et al

The investigation of autophagy modulation as a therapeutic strategy in glia is constrained by an incomplete understanding of the ATG proteins and pathways involved in development and neurodegenerative diseases