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We previously demonstrated that PFOS-induced ROS can result in the autophagy-associated apoptosis in RTCs, which can be suppressed by NAC (a ROS scavenger) through the suppression of PFOS-induced ERK1/2 phosphorylation

95% CI 1.61-2.61) and all-cause mortality (RR = 3.42

Meanwhile, in our opinion, programmed cell death, particularly apoptosis, could be added to this axis because the mechanisms of apoptosis and other forms of programmed cell death are inextricably linked to the mitochondria, mTOR, and autophagy