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The so-called nerve gases and many substances used in insecticides have been shown to act by combining with a residue of serine in the active site of acetylcholine esterase, inhibiting the enzyme completely

Such strategies include the use of AMPK activators (e.g., metformin), mitochondria-targeted antioxidants to mitigate oxidative damage, and nutritional interventions that reactivate autophagic flux

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Additionally, a potential drug binding site can also be predicted based on previous mutagenesis studies that allowed the identification of residues implicated in the transport of doxorubicin, vincristine, and etoposide