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A Unifying Hypothesis of the Pathophysiology of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): Recognitions from the finding of autoantibodies against 2-adrenergic receptors

Therefore, the transfer of the -glutamyl moiety to other amino acids rather than the release of Glu could be advantageous as it can prevent the excitatory cytotoxicity of extracellular Glunotably in injured brain tissue

Oral peptides , though most peptides break down in the digestive system

this has contributed to acquiring a better physiological understanding of their metabolism in humans, while also highlighting the challenges and limitations of the current trials