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The pioneering work of Langers group in the 1980s led to the development of localized controlled-release therapies for GB, culminating in the FDA approval of the first implantable intra-cavity wafer, Gliadel, in 1996 (142)

[DOI] [PMC free article] [PubMed] [Google Scholar] Hodge RD, DErcole AJ, OKusky JR

Oxidative DNA damage Reactive oxygen species, especially OH radical generated during oxidative stress, can react with and cause modifications in all the components of the DNA molecule (the purine and pyrimidine bases and deoxyribose sugar backbone), causing damage such as base or sugar lesions, single-strand breaks, double-strand breaks, abasic site formation and DNADNA or DNAprotein cross-links [3,53,75]

In one, multi-clinic, placebo controlled study employing an add-on design (adjunctive therapy), 144 patients who continued to suffer eight or more CPS per 8 weeks during an 8 week period of monotherapy with doses of either carbamazepine or phenytoin sufficient to assure plasma concentrations within the "therapeutic range" were randomized to receive, in addition to their original antiepilepsy drug (AED), either divalproex sodium or placebo