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Therefore, we asked whether the effect of B 12 on reprogramming could be recapitulated in vitro

10.3390/ijms18051018 44 JaeschkeH.AdelusiO

As the sole intracellular LPO-reducing enzyme, the lack of GPX4 results in excessive accumulation of LPO, destabilizing biological membranes, exacerbating oxidative stress, and ultimately promoting ferroptosis (Ursini and Maiorino, 2020)

This includes the key transcription factor nuclear factor kappa B (NF-B), which triggers the infiltration of inflammatory cells in the pancreas and systemic inflammatory response, leading to acinar cell death through apoptosis and necrosis (9)