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found that different mutations in carcinogenic genes lead to differences in metabolic reprogramming in liver cancer, with the glucose uptake and catabolic metabolism of MYC-induced liver tumors significantly higher than those of MET-induced tumors (83)

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This work was supported by grants from the Postgraduate Education Innovation program of Shanxi Province (2024SJ155, YW), the central government guiding local funds for scientific and technological development (YDZJSX20231A062, CX), scientific and technological achievements transformation guidance project of Shanxi Province (202204021301067, CX), the National Natural Science Foundation of China (82350710800, 82374470, JX), National Key R&D Program of China (2024YFA0919200, JX), Shenzhen Science and Technology Program (KCXFZ20240903094059020, JX), and Shenzhen Medical Research Fund (B2302005, JX)

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