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About this chapter Cite this chapter Nakayama, N

Androgen receptor null male mice develop late-onset obesity caused by decreased energy expenditure and lipolytic activity but show normal insulin sensitivity with high adiponectin secretion

They went on to suggest that the therapeutic properties of ALCAR supplementation is a viable therapy for brain regeneration .[iv] ALCAR promotes brain energy metabolism by supporting your brain cells mitochondria in creating ATP which is your main source of cellular energy.[v] ALCAR works as a shuttle transport for fatty acids through cell membranes into mitochondria to assist with adenosine triphosphate (ATP) synthesis .[vi] ALCAR is essential for fatty acid oxidation: ALCAR shuttles fatty acids into mitochondria, for the creation of ATP