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However, the mechanism(s) of increased blood serotonin (5-hydroxytryptamine, 5-HT) in a subset of individuals with ASD remain(s) not clearly elucidated, although serotonin metabolism (Fig

We also demonstrated that the locomotor function of mdx mice was maintained for a long time after DPSC treatment ( mdx mice ( Choline-containing compounds were approximately three times higher than those in healthy individuals and patients with other myopathies, whereas creatine levels were within the normal range, indicating that abnormal cell membrane function may be correlated with abnormal dystrophin or lack of dystrophin in the brains of patients with DMD ( mdx mice was similar to that in patients with DMD and was downregulated after DPSC-treatment (Figure 4B)

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This pathological process, characterized by initial ischemia followed by reperfusion, paradoxically exacerbates cellular and tissue damage, leading to acute kidney injury (AKI) and compromising graft survival and patient outcomes