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Recent studies have shown that, beyond eliminating pathogens through traditional phagocytosis, neutrophils participate in anti-infective defense and inflammatory responses by releasing neutrophil extracellular traps (NETs), secreting inflammatory mediators (e.g., cytokines, chemokines, and lipid mediators), and producing ROS (103, 104)

The ultimate goal of the research is to develop mitochondrial-targeted therapies that can intelligently respond to pathological states and to achieve stage-based individualized diagnosis and treatment of sepsis using mitochondrial function markers, thereby systematically innovating the existing clinical prevention and treatment strategies