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GH is produced and distributed by the pituitary gland, the part of the body that is responsible for controlling and maintaining the endocrine system

(2008) demonstrated that systemic administration of IGF-1 LR3 to dystrophic mdx mice (a model of Duchenne muscular dystrophy) significantly reduced susceptibility to contraction-mediated damage in extensor digitorum longus, soleus, and diaphragm muscles

The Teichman human study reported sustained, dose-dependent elevation of both GH and IGF-I for days after a single CJC-1295-with-DAC dose, [2] and broader work confirms that CJC-1295 activates the GH/IGF-1 axis in healthy human subjects via pituitary GHRH-receptor binding