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GHK appears to directly target myofibroblasts, converting pathological persistent collagen deposition to physiological collagen contraction through ITG-1 activation and apoptotic elimination of excess myofibroblasts (Figure 5)

While the molecular alterations identified were robust and internally consistent, validation in larger, independent cohorts will be important to confirm our findings and assess inter-individual variability

Tissue Regeneration and Organ Health Clearing senescent cells creates space for healthy progenitor cells to proliferate and replace damaged tissue