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The aetiological agents of CD are gluten immunogenic peptides (GIPs) derived from Triticeae prolamins (Rodrguez-Ramrez et al, 2024), which, unlike most food proteins, are not fully digested by the peptidases from the gastrointestinal tract (GIT) due to their high proline content (roughly 35%) (Bonner et al, 2025)

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